k Multiple sclerosis. Coloured Scanning Electron Micrograph SEM of microglial cells yellow ingesting branched oligodendrocyte cells purple. This is the process thought to occur in multiple sclerosis MS. Oligodendrocytes form insulating myelin sheaths around nerve axons in the central nervous system. Microglia normally ingest cell debris and bacteria as part of the bodys immune response. In MS they attack oligodendrocytes, possibly triggered by a virus in people with hereditary susceptibility. Destruction of myelin sheaths leads to loss of nerve function. Symptoms include unsteady gait, visual speech defects and paralysis. Magnification x875 at 5x7cm size. Editorial Stock Photo - Afloimages
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Multiple sclerosis. Coloured Scanning Electron Micrograph (SEM) of microglial cells (yellow) ingesting branched oligodendrocyte cells (purple). This is the process thought to occur in multiple sclerosis (MS). Oligodendrocytes form insulating myelin sheaths around nerve axons in the central nervous system. Microglia normally ingest cell debris and bacteria as part of the body's immune response. In MS they attack oligodendrocytes, possibly triggered by a virus in people with hereditary susceptibility. Destruction of myelin sheaths leads to loss of nerve function. Symptoms include unsteady gait, visual & speech defects and paralysis. Magnification: x875 at 5x7cm size.
ED

Multiple sclerosis. Coloured Scanning Electron Micrograph (SEM) of microglial cells (yellow) ingesting branched oligodendrocyte cells (purple). This is the process thought to occur in multiple sclerosis (MS). Oligodendrocytes form insulating myelin sheaths around nerve axons in the central nervous system. Microglia normally ingest cell debris and bacteria as part of the body's immune response. In MS they attack oligodendrocytes, possibly triggered by a virus in people with hereditary susceptibility. Destruction of myelin sheaths leads to loss of nerve function. Symptoms include unsteady gait, visual & speech defects and paralysis. Magnification: x875 at 5x7cm size.

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ID
10594925

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License type
Editorial

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Creation date
17-11-2010

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